Pulmonary vascular permeability changes in an ovine model of methicillin-resistant Staphylococcus aureus sepsis
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Date
2009-02
Authors
Jonkam, C. C.
Bansal, K.
Traber, D. L.
Hamahata, A.
Maybauer, M. O.
Maybauer, D. M.
Cox, R. A.
Lange, M.
Connelly, R. L.
Traber, L. D.
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BioMed Central
Abstract
Endothelial dysfunction is a hallmark of sepsis, associated with lung transvascular fluid flux and pulmonary dysfunction in septic patients. We tested the hypothesis that methicillin-resistant Staphylococcus aureus (MRSA) sepsis following smoke inhalation increases pulmonary transvascular fluid flux via excessive nitric oxide (NO) production.
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Citation
C.C. Jonkam, K. Bansal, D.L. Traber, A. Hamahata, M.O. Maybauer, D.M. Maybauer, R.A. Cox, M. Lange, R.L. Connelly, L.D. Traber, C.D. Djukom, J.R. Salsbury, D.N. Herndon, and P. Enkhbaatar, “Pulmonary vascular permeability changes in an ovine model of methicillin-resistant Staphylococcus aureus sepsis,” Critical Care 2009, 13:R19, Feb. 17, 2009.